CYCLOFENIL (SAME CHEMICAL NAME)
Produced By:|
Reported Characteristics
Cyclofenil administration is quite similar to using HCG and CLOMID together. Itis an anti-estrogen and a gonadotropin/testosterone stimulator. It does so by occupyingestrogen receptors (antagonist) with a much weaker estrogenic compound and by shuttingdown the negative feed-back within the hypothalamus-pituitary-testes-axis (HPTA). Themale body actually can produce much more testosterone than it does. Simply said, it seesno reason to do so. When the hypothalamus senses adequate testosterone/estrogen levels,either naturally (endogenous) or unnaturally (exogenous) provided, it shuts offgonadotropin release from the pituitary/hypothalamus. Thus, leydig cells in the testes donot receive the signal to produce more testosterone. Cyclofenil interferes with thisnegative feed-back/shut down production signal. This means that the hypothalamus -pituitary-testes-axis runs wide open to some extent, and more testosterone is produced.After about 5-6 weeks the hypothalamus figures this out and really shuts down thegoodies production. Though some strength and mass resulted with Cyclofenil use, it was not noted asa great growth drug. Some "natural" (okay) bodybuilders have used it with some resultsas have older individuals. However, the chemically assisted lads have used it to kick startnatural testosterone production and even as a "during cycle anti-estrogen" verysuccessfully. Normally 200 mg was taken 2-3 times daily for 4-6 weeks, either startingthe last 3 weeks prior to the end of an AAS cycle or directly following a cycle. The priormethod being reported as more effective since about a week is necessary for Cyclofenil tobecome effective and provide results. Side effects commonly reported have been; lightacne, elevated sex-drive (that is a side effect?), and hot flashes. TRADE NAMES
*A note of interest; I have known several so-called natural bodybuilders who haveutilized cyclofenil with a prohormone protocols in the United States with surprising results.This was effective for many reasons. Lower estrogen activity means a harder appearance.Testosterone both endogenous and exogenous combine resulted in significant totaltestosterone levels as well as a higher free testosterone level due to prohormones ability touncouple testosterone from SHGB to some extent. The usual cycle consisted of 6 weeks ofprohormones with 4 weeks of cyclofenil beginning week #5 of the prohormone cycle. Doingso extended the cycle 2 weeks and assured no post-cycle HPTA suppression to deal with formost athletes. Some athletes reported week #6-8 produced good secondary growth. Thismay have been due to an up- regulation of LH, FSH, and the conversion enzymesresponsible for prohormones becoming testosterone in blood as well as decreased activity ofestrogen. My concern would be the rush of estrogen post-cycle since cyclofenil only blocksestrogen receptors instead of suppressing estrogen production. In that example, for mepersonally, Arimidex would have been a wiser choice beginning week #5 also. |
